7. Ngo, S.T., Steyn, F.J. and McCombe, P.A. (2014). Gender differences in autoimmune disease. Frontiers in Neuroendocrinology
35: 347–369.8. Krolewski, A.S. et al. (1987). Epidemiologic approach to the etiology of type 1 diabetes mellitus and its complications. The New England Journal of Medicine
26: 1390–1398.9. Bach, J.-F. (2002). The effect of infections on susceptibility to autoimmune and allergic diseases. The New England Journal of Medicine
347: 911–920.10. Uramoto, K.M. et al. (1999) Trends in the incidence and mortality of systemic lupus erythematosus, 1950–1992. Arthritis & Rheumatism
42: 46–50.11. Alonso, A. and Hernan, M.A. (2008). Temporal trends in the incidence of multiple sclerosis: A systematic review. Neurology
71: 129–135.12. Werner, S. et al. (2002). The incidence of atopic dermatitis in school entrants is associated with individual lifestyle factors but not with local environmental factors in Hannover, Germany. British Journal of Dermatology
147: 95–104.Глава 2
1. Bairlein, F. (2002). How to get fat: nutritional mechanisms of seasonal fat accumulation in migratory songbirds. Naturwissenschaften
89: 1–10. 2. Heini, A.F. and Weinsier, R.L. (1997). Divergent trends in obesity and fat intake patterns: The American paradox. American Journal of Medicine 102: 259–264.3. Silventoinen, K. et al.
(2004). Trends in obesity and energy supply in the WHO MONICA Project. International Journal of Obesity 28: 710–718.4. Troiano, R.P. et al
. (2000). Energy and fat intakes of children and adolescents in the United States: data from the National Health and Nutrition Examination Surveys. American Journal of Clinical Nutrition 72: 1343s–1353s.5. Prentice, A.M. and Jebb, S.A. (1995). Obesity in Britain: Gluttony or sloth? British Journal of Medicine
311: 437–439.6. Westerterp, K.R. and Speakman, J.R. (2008). Physical activity energy expenditure has not declined since the 1980s and matches energy expenditures of wild mammals. International Journal of Obesity
32: 1256–1263.7. World Health Organisation (2014). Global Health Observatory Data – Overweight and Obesity. Available at: http://www.who.int/gho/ncd/risk_factors/overweight/en/.
8. Speliotes, E.K. et al.
(2010). Association analyses of 249,796 individuals reveal 18 new loci associated with body mass index. Nature Genetics 42: 937–948.9. Marshall, J.K. et al.
(2010). Eight year prognosis of postinfectious irritable bowel syndrome following waterborne bacterial dysentery. Gut 59: 605–611.10. Gwee, K.-A. (2005). Irritable bowel syndrome in developing countries – a disorder of civilization or colonization? Neurogastroenterology and Motility
17: 317–324.11. Collins, S.M. (2014). A role for the gut microbiota in IBS. Nature Reviews Gastroenterology and Hepatology
11: 497–505.12. Jeffery, I.B. et al
. (2012). An irritable bowel syndrome subtype defined by species-specific alterations in faecal microbiota. Gut 61: 997–1006.13. Backhed, F. et al.
(2004). The gut microbiota as an environmental factor that regulates fat storage. Proceedings of the National Academy of Sciences 101: 15718–15723.14. Ley, R.E. et al
. (2005). Obesity alters gut microbial ecology. Proceedings of the National Academy of Sciences 102: 11070–11075.15. Turnbaugh, P.J. et al
. (2006). An obesity-associated gut microbiome with increased capacity for energy harvest. Nature 444: 1027–1031.16. Centers for Disease Control (2014). Obesity Prevalence Maps. Available at: http://www.cdc.gov/obesity/data/prevalence-maps.html.
17. Gallos, L.K. et al. (2012). Collective behavior in the spatial spreading of obesity. Scientific Reports
2: no. 454.18. Christakis, N.A. and Fowler, J.H. (2007). The spread of obesity in a large social network over 32 years. The New England Journal of Medicine
357: 370–379.19. Dhurandhar, N.V. et al.
(1997). Association of adenovirus infection with human obesity. Obesity Research 5: 464–469.20. Atkinson, R.L. et al.
(2005). Human adenovirus-36 is associated with increased body weight and paradoxical reduction of serum lipids. International Journal of Obesity 29: 281–286.21. Everard, A. et al.
(2013). Cross-talk between Akkermansia muciniphila and intestinal epithelium controls diet-induced obesity. Proceedings of the National Academy of Sciences 110: 9066–9071.