Энергия мозга. Теория развития всех психических заболеваний, объясняющая их общую причину

3. D. C. Wallace. “A Mitochondrial Etiology of Neuropsychiatric Disorders.” JAMA Psychiatry 74(9) (2017): 863–864. doi: 10.1001/jamapsychiatry.2017.0397.

4. T. Kozicz, A. Schene, and E. Morava. “Mitochondrial Etiology of Psychiatric Disorders: Is This the Full Story?” JAMA Psychiatry 75(5) (2018): 527. doi: 10.1001/jamapsychiatry.2018.0018.

5. M. D. Brand and D. G. Nicholls. “Assessing Mitochondrial Dysfunction in Cells [published correction appears in Biochem J 437(3) (August 1, 2011): 575]. Biochem J 435(2) (2011): 297–312. doi: 10.1042/ BJ20110162.

6. I. R. Lanza and K. S. Nair. “Mitochondrial Metabolic Function Assessed In Vivo and In Vitro.” Curr Opin Clin Nutr Metab Care 13(5) (2010): 511–517. doi: 10.1097/MCO.0b013e32833cc93d.

7. A. H. De Mello, A. B. Costa, J. D. G. Engel, and G. T. Rezin. “Mitochondrial Dysfunction in Obesity.” Life Sci 192 (2018): 26–32. doi: 10.1016/j.lfs.2017.11.019.

8. P. H. Reddy and M. F. Beal. “Amyloid Beta, Mitochondrial Dysfunction and Synaptic Damage: Implications for Cognitive Decline in Aging and Alzheimer’s Disease.” Trends Mol Med 14(2) (2008): 45–53. doi: 10.1016/j.molmed.2007.12.002.

9. Estela Area-Gomez, Ad de Groof, Eduardo Bonilla, Jorge Montesinos, Kurenai Tanji, Istvan Boldogh, Liza Pon, and Eric A. Schon. “A Key Role for MAM in Mediating Mitochondrial Dysfunction in Alzheimer Disease.” Cell Death Dis 9(3) (2018): 335. doi: 10.1038/s41419-017-0215-0; R. H. Swerdlow. “Mitochondria and Mitochondrial Cascades in Alzheimer’s Disease.” J Alzheimers Dis 62(3) (2018): 1403–1416. doi: 10.3233/JAD-170585.

10. Fei Du, Xiao-Hong Zhu, Yi Zhang, Michael Friedman, Nanyin Zhang, Kâmil Uğurbil, and Wei Chen. “Tightly Coupled Brain Activity and Cerebral ATP Metabolic Rate.” Proc Natl Acad Sci USA 105(17) (2008): 6409. doi: 10.1073/pnas.0710766105.

11. K. Todkar, H. S. Ilamathi, M. Germain. “Mitochondria and Lysosomes: Discovering Bonds.” Front Cell Dev Biol 5 (2017):106. doi: 10.3389/fcell.2017.00106.

12. Q. Chu, T. F. Martinez, S. W. Novak, et al. “Regulation of the ER Stress Response by a MITOCHONDRIAL MICROPROTEIN.” Nat Commun 10 (2019): 4883. doi: 10.1038/s41467-019-12816-z.

13. B. Kalman, F. D. Lublin, and H. Alder. “Impairment of Central and Peripheral Myelin in Mitochondrial Diseases.” Mult Scler 2(6) (1997): 267–278. doi: 10.1177/135245859700200602; E. M. R. Lake, E. A. Steffler, C. D. Rowley, et al. “Altered Intracortical Myelin Staining in the Dorsolateral Prefrontal Cortex in Severe Mental Illness.” Eur Arch Psychiatry Clin Neurosci 267 (2017): 369–376. doi: 10.1007/s00406-0160730-5; J. Rice and C. Gu. “Function and Mechanism of Myelin Regulation in Alcohol Abuse and Alcoholism.” Bioessays 41(7) (2019): e1800255. doi: 10.1002/bies.201800255. Epub May 16, 2019; Gerhard S. Drenthen, Walter H. Backes, Albert P. Aldenkamp, R. Jeroen Vermeulen, Sylvia Klinkenberg, and Jacobus F. A. Jansen. “On the Merits of Non-Invasive Myelin Imaging in Epilepsy, a Literature Review.” J Neurosci Methods 338 (2020): 108687. doi: 10.1016/j.jneumeth.2020.108687; E. Papuć and K. Rejdak. “The Role of Myelin Damage in Alzheimer’s Disease Pathology.” Arch Med Sci 16(2) (2018): 345–351. doi: 10.5114/ aoms.2018.76863; G. Cermenati, F. Abbiati, S. Cermenati, et al. “Diabetes-Induced Myelin Abnormalities Are Associated with an Altered Lipid Pattern: Protective Effects of LXR Activation.” J Lipid Res 53(2) (2012): 300–310. doi: 10.1194/jlr.M021188; M. Bouhrara, N. Khattar, P. Elango, et al. “Evidence of Association Between Obesity and Lower Cerebral Myelin Content in Cognitively Unimpaired Adults.” Int J Obes (Lond) 45(4) (2021): 850–859. doi: 10.1038/s41366-021-00749-x.

14. A. Ebneth, R. Godemann, K. Stamer, S. Illenberger, B. Trinczek, and E. Mandelkow. “Overexpression of Tau Protein Inhibits Kinesin-Dependent Trafficking of Vesicles, Mitochondria, and Endoplasmic Reticulum: Implications for Alzheimer’s Disease.” J Cell Biol 143(3) (1998): 777–794. doi: 10.1083/jcb.143.3.777.

15. A. Cheng, J. Wang, N. Ghena, Q. Zhao, I. Perone, T. M. King, R. L. Veech, M. Gorospe, R. Wan, and M. P. Mattson. “SIRT3 Haploinsufficiency Aggravates Loss of GABAergic Interneurons and Neuronal Network Hyperexcitability in an Alzheimer’s Disease Model.” J Neurosci 40(3) (2020): 694–709. doi: 10.1523/ JNEUROSCI.1446-19.2019.

16. J. Mertens, et al. “Differential Responses to Lithium in Hyperexcitable Neurons from Patients with Bipolar Disorder.” Nature 527(7576) (2015): 95–99. doi: 10.1038/nature15526.

17. J. A. Rosenkranz, E. R. Venheim, and M. Padival. “Chronic Stress Causes Amygdala Hyperexcitability in Rodents.” Biol Psychiatry 67(12) (2010): 1128–1136. doi: 10.1016/j.biopsych.2010.02.008.

18. Marco Morsch, Rowan Radford, Albert Lee, Emily Don, Andrew Badrock, Thomas Hall, Nicholas Cole, and Roger Chung. “In Vivo Characterization of Microglial Engulfment of Dying Neurons in the Zebrafish Spinal Cord.” Front Cell Neurosci 9 (2015): 321. doi: 10.3389/fncel.2015.00321.