Клетка «на диете». Научное открытие о влиянии жиров на мышление, физическую активность и обмен веществ

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12. K. J. Bough et al., “Mitochondrial Biogenesis in the Anticonvulsant Mechanism of the Ketogenic Diet,” Annals of Neurology, 60 (2006): 223-35, D01:10.1002/ana.20899.

13. P. J. Cox, K. Clarke, “Acute Nutritional Ketosis: Implications for Exercise Performance and Metabolism,” Extreme Physiology & Medicine, 3 (2014): 1, DOI: 10.1186/2046-7648-3-17.

14. О. E. Owen et al., “Liver and Kidney Metabolism During Prolonged Starvation,” Journal of Clinical Investigation, 48, no. 3 (1969): 574-83.

15. M. Akram, “A Focused Review of the Role of Ketone Bodies in Health and Disease,” Journal of Medicinal Food, 16, no. 11 (November 2013): 965-67, DOI: 10.1089/jmf.2012.2592.

16. Там же.

17. Phinney and Volek, The Art and Science of Low-Carbohydrate Living, 10.

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20. A. Paoli et al., “Ketogenic Diet in Neuromuscular and Neurodegenerative Diseases,” BioMed Research International, 2014 (2014), DOL10.1155/2014/474296.

21. M. A. McNally and A. L. Hartman, “Ketone Bodies in Epilepsy,” Journal of Neurochemistry, 121, no. 1 (2012): 28–35, DOI: 10.1111/j.l471-4159.2012.07670.x.

22. J. Moore, Keto Clarity (Victory Belt Publishing, 2014), 58.

23. A. J. Brown, “Low-Carb Diets, Fasting and Euphoria: Is There a Link between Ketosis and Gamma-hydroxybutyrate (GHB)?” Medical Hypotheses, 68, no. 2 (2007): 268-71, DOI: 10.1016/j.mehy.2006.07.043.

Глава 3

1. E. L. Knight et al., “The Impact of Protein Intake on Renal Function Decline in Women with Normal Renal Function or Mild Renal Insufficiency,” Annals of Internal Medicine, 138. no. 6 (2003): 460-67, DOI: 10.7326/0003-4819-138-6-200303180-00009.

2. M. I. Frisard et al., “Effect of 6-Month Calorie Restriction on Biomarkers of Longevity, Metabolic Adaptation, and Oxidative Stress in Overweight Individuals: A Randomized Controlled Trial,” http://jamanetwork.com/journals/jama/fullarti-cle/1108368.

3. M. E. Levine et al., “Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population,” Cell Metabolism, 19, no. 3 (2014): 407-17, DOI: 10.1016/j. cmet.2014.02.006.

4. J. Guevara-Aguirre et al., “Growth Hormone Receptor Deficiency Is Associated With a Major Reduction in Pro-aging Signaling, Cancer and Diabetes in Humans,” Science Translational Medicine, 3, no. 70 (2011): 70, DOI: 10.1126/ scitranslmed.3001845.

5. S. I. A. Apelo and D. W. Lamming, “Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island,” Journal of Gerontology, 71, no. 7 (2016): 841–849, DOI: 10.1093/gerona/glw090.

6. S. M. Solon-Biet et al., “The Ratio of Macronutrients, Not Caloric Intake, Dictates Cardiometabolic Health, Aging, and Longevity in Ad Libi-tum-Fed Mice,” Cell Metabolism, 19, no. 3 (2014): 418-30, DOI: 10.1016/j. cmet.2014.02.009.

Глава 4

1. “Ferritin: The Test,” American Association for Clinical Chemistry, https://labtest-sonline.org/understanding/analytes/ferritin/tab/test/, accessed May 9, 2016.

2. E. D. Weinberg, “The Hazards of Iron Loading,” Metallomics, 2, no. 11 (November, 2010):732-40, DOI: 10.1039/c0mt00023j.

3. M. D. Beaton and P. C. Adams, “Treatment of Hyperferritinemia,” Annals of Hepatology, 11, no. 3 (2012): 294–300, PMID: 22481446.

4. G. Ortiz-Estrada et al., “Iron-Saturated Lactoferrin and Pathogenic Protozoa: Could This Protein Be an Iron Source for Their Parasitic Style of Life?” Future Microbiology, 7, no. 1 (2012): 149-64, DOI: 10.2217/fmb. 11.140.

5. D. J. Fleming et al., “Dietary Factors Associated with the Risk of High Iron Stores in the Elderly Framingham Heart Study Cohort,” American Journal of Clinical Nutrition, 76, no. 6 (2002): 1375-84, PMID: 12450906.

6. T. Iwasaki et al., “Serum Ferritin Is Associated with Visceral Fat Area and Subcutaneous Fat Area,” Diabetes Care, 28, no. 10 ("2005): 2486-91, PMID: 16186284.