Клеточные и молекулярные механизмы патогенеза иммуновоспалительных ревматических заболеваний

96. Loo J., Spittle D. A., Newnham M. COVID-19, immunothrombosis and venous thromboembolism: biological mechanisms. Thorax Published Online First: 06 January 2021. doi: 10.1136/thoraxjnl-2020–216243.

97. Ma W-T., Gao F., Gu K., Chen D-K. The Role of Monocytes and Macrophages in Autoimmune Diseases: A Comprehensive Review. Front. Immunol., 2019; 10:1140. doi: 10.3389/fimmu.2019.01140.

98. Malmstrom V., Venalis P., Albrecht I. T cells in myositis. Arthritis Res. Ther., 2012; 14(6), 230. doi.org/10.1186/ar4116.

99. Mantovani A., Sozzani S., Locati M., Allavena P., Sica A. Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes. Trends Immunol., 2002, Vol. 23, no.11, pp.549–555. doi: 10.1016/s1471–4906(02)02302–5.

100. Manzo A., Bombardieri M., Humby F., Pitzalis C. Secondary and ectopic lymphoid tissue responses in rheumatoid arthritis: from inflammation to autoimmunity and tissue damage/remodeling. Immunol Rev., 2010, Vol. 233, pp.267–285. doi: 10.1111/j.0105–2896.2009.00861.x.

101. Masters S. L., Simon A., Aksentijevich I., Kastner D. L. Horror Autoinflammaticus: The Molecular Pathophysiology of Autoinflammatory Disease. Annu. Rev. Immunol., 2009, Vol. 27, pp.621–668. doi: 10.1146/annurev.immunol.25.022106.141627.

102. McNally A. K., Anderson J. M. Interleukin-4 induces foreign body giant cells from human monocytes/macrophages. Differential lymphokine regulation of macrophage fusion leads to morphological variants of multinucleated giant cells. Am. J. of Pathology, 1995, Vol. 147, no.5, pp. 1487–1499.

103. McNally A. K., Jones J. A., Macewan S. R., Colton E., Anderson J. M. Vitronectin is a critical protein adhesion substrate for IL-4-induced foreign body giant cell formation. Journal of Biomedical Materials Research, 2008, Vol. 86, no. 2, pp. 535–543. doi: 10.1002/jbm.a.31658.

104. Means T. K., Latz E., Hayashi F., Murali M. R., Golenbock D. T., Luster A. D. Human lupus autoantibody-DNA complexes activate DCs through cooperation of CD32 and TLR9. J. Clin. Investig., 2005, Vol. 115, no. 2, pp. 407–417. doi: 10.1172/JCI23025.

105. Miga A., Masters S., Gonzalez M., Noelle R. J. The role of CD40-CD154 interactions in the regulation of cell mediated immunity. Immunological Investigations, 2000, Vol.29, no 2, pp. 111–114. doi: 10.3109/08820130009062292.

106. Miyabe Y., Lian, J., Miyabe, C., Luster, A. D. Chemokines in rheumatic diseases: pathogenic role and therapeutic implications. Nature Reviews Rheumatology, 2019, 15:731–46. doi:10.1038/s41584–019–0323–6.

107. Moghaddas F., Masters S. L. Monogenic autoinflammatory diseases: cytokinopathies. Cytokine, 2015, Vol. 74, no.2, pp.237–246. doi: 10.1016/j.cyto.2015.02.012.

108. Moore B. B., Keane M. P., Addison C. L., Arenberg D. A., Strieter R. M., CXC chemokine modulation of angiogenesis: the importance of balance between angiogenic and angiostatic members of the family. J. Invest. Med., 1998, Vol. 46, p. 113.

109. Murphy G., Knauper V., Atkinson S., Butler G., English W, Hutton M., Stracke J., Clark I. Matrix metalloproteinases in arthritic disease. Arthritis Res., 2002, 4(Suppl 3):S39–S49. doi: 10.1186/ar572.

110. Murshid A., Gong J., Calderwood S. K. The role of heat shock proteins in antigen cross presentation. Front. Immunol., 2012, Vol.3, Article63. doi: 10.3389/fimmu.2012.00063. eCollection.

111. Nakhasi H. L., Ramanujam M., Atreya C. D., Hobman T. C., Lee N. Rubella virus glycoprotein interaction with the endoplasmic reticulum calreticulin and calnexin. Arch. Virol., 2001, Vol. 146, pp.1–14.

112. Nanki T., Hayashida K., El-Gabalawy H., Suson S., Shi K., Girschick H. J., Yavus S., Lipsky P. E. Stromal cell-derived factor-1-CXC chemokine receptor 4 interactions play a central role in CD4+ T-cell accumulation in rheumatoid arthritis synovium. J Immunol., 2000, Vol. 165, no. 11, pp. 6590–6598. doi: 10.4049/jimmunol.165.11.6590.

113. Nanki T., Shimaoka T., Hayashida K., Taniguchi K., Yonehara S., Miyasaka N.. Pathogenic role of the CXCL16-CXCR6 pathway in rheumatoid arthritis. Arthritis Rheum., 2005, Vol. 52, no.10, pp. 3004–3014. doi: 10.1002/art.21301.