Невидимая угроза. Как Wi-Fi, сотовые телефоны, 5G и другие электромагнитные излучения влияют на наше самочувствие и продолжительность жизни

126. Chen H, Qu Z, Liu W. “Effects of Simulated Mobile Phone Electromagnetic Radiation on Fertilization and Embryo Development.” Fetal and Pediatric Pathology. Vol. 36, no. 2. (April 2017): 123–129. doi: 10.1080/15513815.2016.1261974.

Глава 6. Как восстановить повреждения, нанесенные ЭМП

1. Hopp AK, Grüter P, Hottiger MO. “Regulation of Glucose Metabolism by NAD+ and ADP-Ribosylation.” Cells.Vol. 8, no. 8. (August 2019): 890. doi: 10.3390/cells8080890.

2. Virág L, Szabo C. “The Therapeutic Potential of Poly(ADP-ribose) Polymerase Inhibitors.” Pharmacological Reviews. Vol. 54, no. 3. (September 2002): 375–429.

3. Shall S, de Murcia G. “Poly(ADP-ribose) Polymerase-1: What Have We Learned from the Deficient Mouse Model?” Mutation Research. Vol. 460, no. 1. (June 30, 2000): 1–15.

4. Alemasova EE, Lavrik OI. “Poly(ADP-ribosyl)ation by PARP1: Reaction Mechanism and Regulatory Proteins.” Nucleic Acids Research. Vol. 47, no. 8. (February 25, 2019): 3811–3827. doi: 10.1093/nar/gkz120.

5. Schraufstatter IU, Hinshaw DB, Hyslop PA, Spragg RG, Cochrane CG. “Oxidant Injury of Cells. DNA Strand-Breaks Activate Polyadenosine Diphosphate-Ribose Polymerase and Lead to Depletion of Nicotinamide Adenine Dinucleotide.” Journal of Clinical Investigation. Vol. 77, no. 4. (April 1, 1986): 1312–1320. doi: 10.1172/JCI112436.

6. Bai P. “PARP-1 Inhibition Increases Mitochondrial Metabolism through SIRT1 Activation.” Cell Metabolism.Vol. 13, no. 4. (April 6, 2011): 461–446.

7. Pirinen E, Cantó C, Jo YS, Morato L, Zhang H, Menzies KJ, Williams EG, Mouchiroud L, Moullan N, Hagberg C, Li W, Timmers S, Imhof R, Verbeek J, Pujol A, van Loon B, Viscomi C, Zeviani M, Schrauwen P, Sauve AA, Schoonjans K, Auwerx J. “Pharmacological Inhibition of Poly(ADP-Ribose) Polymerases Improves Fitness and Mitochondrial Function in Skeletal Muscle.” Cell Metabolism. Vol. 19, no. 6. (June 3, 2014): 1034–1041. doi: 10.1016/j.cmet.2014.04.002.

8. Massudi H, Grant R, Braidy N, Guest J, Farnsworth B, Guillemin GJ. “AgeAssociated Changes in Oxidative Stress and NAD+ Metabolism in Human Tissue.” PLoS One. Vol. 7, no. 7. (2012): e42357.

9. Braidy N, Guillemin GJ, Mansour H, Chan-Ling T, Poljak A, Grant R. “Age Related Changes in NAD+ Metabolism, Oxidative Stress, and Sirt1 Activity in Wistar Rats.” PLoS One. Vol. 6, no. 4. (April 26, 2011): e19194.

10. Makvandi M, Sellmyer MA, Mach RH. “Inflammation and DNA Damage: Probing Pathways to Cancer and Neurodegeneration.” Drug Discovery Today: Technologies. Vol. 25. (November 2017): 37–43. doi: 10.1016/j.ddtec.2017.11.001.

11. Berger, F. “The New Life of a Centenarian: Signalling Functions of NAD(P).” Trends in Biochemical Sciences. Vol. 29, no. 3. (2004): 111–118. doi: 10.1016/j.tibs.2004.01.007.

12. Warburg O, Pyridine CW. “Pyridine, the Hydrogen Transfusing Component of Fermentative Enzymes.” Helvetica Chimica Acta. Vol. 19. (1936): 79–88.

13. Sinclair DA, Guarente L. “Unlocking the Secrets of Longevity Genes.” Scientific American. Vol. 294, no. 3. (March 2006): 48–51, 54–57.

14. Romani M. “Niacin: An Old Lipid Drug in a New NAD+ Dress.” Journal of Lipid Research. Vol. 60, no. 4. (April 2019): 741–746. doi: 10.1194/jlr.S092007.

15. Braidy N, Berg J, Clement J, Khorshidi F, Poliak A, Javasena T, Grant R, Sachdev P. “Role of NAD+ and Related Precursors as Therapeutic Targets for Age-Related Degenerative Diseases: Rationale, Biochemistry, Pharmacokinetics, and Outcomes.” Antioxidants & Redox Signal. Vol. 30, no. 2. (January 10, 2019): 251–294. doi: 10.1089/ars.2017.7269.

16. Ansari HR, Raghava GP. “Identification of NAD Interacting Residues in Proteins.” BMC Bioinformatics. Vol. 11. (March 30, 2010): 160.

17. Placzek S, Schomburg I, Chang A, Jeske L, Ulbrich M, Tillack J, Schomburg D. “BRENDA in 2017: New Perspectives and New Tools in BRENDA.” Nucleic Acids Research. Vol. 45. (January 4, 2017): D380–D388.

18. Conze D, Brenner C, Kruger CL. “Safety and Metabolism of Long-Term Administration of NIAGEN (Nicotinamide Riboside Chloride) in a Randomized, Double-Blind, Placebo-Controlled Clinical Trial of Healthy Overweight Adults.” Scientific Reports. Vol. 9, no. 1. (July 5, 2019): 9772. doi: 10.1038/s41598-019-46120-z.

19. Canto C, Menzies KJ, and Auwerx J. “NAD(+) Metabolism and the Control of Energy Homeostasis: A Balancing Act Between Mitochondria and the Nucleus.” Cellular Metabolism. Vol. 22. (2015): 31–53.