Novelli A., et al.
Glutamate becomes neurotoxic via the N-methyl-Daspartate receptor when intracellular energy levels are reduced. Brain Res.1988 Jun 7; 451(1–2):205–12. doi:10.1016/0006-8993(88)90765-2. Ristow M., et al. Frataxin activates mitochondrial energy conversion and oxidative phosphorylation. Proc Natl Acad Sci U S A. 2000; 97(№ 22):12239–43. doi:10.1073 /pnas.220403797.Schon E. A., Manfredi G.
Neuronal degeneration and mitochondrial dysfunction. J Clin Invest. 2003 Feb; 111(3):303–12. doi:10.1172/JCI17741. Schulte E. C., et al. Mitochondrial membrane protein associated neurodegenration: A novel variant of neurodegeneration with brain iron accumulation. Mov Disord. 2013 Feb; 28(2):224–7. Epub 2012 Nov 19. doi:10.1002/mds.25256.Schulz J. B., et al.
Neuroprotective strategies for treatment of lesions produced by mitochondrial toxins: implications for neurodegenerative diseases. Neuroscience 71. 1996; 71(4):1043–48. doi:10.1016/0306-4522(95)00527-7.Sobreira C, et al.
Mitochondrial encephalomyopathy with coenzyme Q10 deficiency. Neurology. 1997 May; 48(5):1238–43.Sun T., et al.
Motile axonal mitochondria contribute to the variability of presynaptic strength. Cell Rep. 2013 Aug 15; 4(3):413–9. Epub 2013 Jul 23. doi:10.1016/j.celrep.2013.06.040.Tatton W. G., Chalmers-Redman R. M.
Mitochondria in neurodegenerative apoptosis: an opportunity for therapy? Ann Neurol. 1998; 44(3 Suppl 1):S. 134–S. 141. doi:10.1002/ana.410440720.Tatton W. G, Olanow C. W.
Apoptosis in neurodegenerative diseases: the role of mitochondria. Biochim Biophys Acta. 1999 Feb 9; 1410(2):195–213. doi:10.1016/S0005-2728(98)00167-4.Turner C., Schapira A. H.
Mitochondrial dysfunction in neurodegenerative disorders and ageing. Adv Exp Med Biol. 2001; 487:229–51.Veitch K. et al.
Global ischemia induces a biphasic response of the mitochondrial respiratory chain. Anoxic pre-perfusion protects against ischaemic damage. Biochem J. 1992 Feb 1; 281(Pt 3):709–15.Volpe M., Cosentino F.
Abnormalities of endothelial function in the pathogenesis of stroke: the importance of endothelin. J Cardiovasc Pharmacol. 2000; 35(4 Suppl 2):S. 45–S. 48.Болезнь Альцгеймера: не забывайте о митохондриях!
Berger A.
The Alzheimer’s antidote: Using a low-carb, high-fat diet to fight Alzheimer’s disease, memory loss, and cognitive decline. White River Junction, VT: Chelsea Green Publishing, 2017.Blass J. P.
The mitochondrial spiral. An adequate cause of dementia in the Alzheimer’s syndrome. Ann NY Acad Sci. 2000; 924:170–83. doi:10.1111/j.1749–6632.2000.tb05576.x.Bonilla E., et al.
Mitochondrial involvement in Alzheimer’s disease. Biochim Biophys Acta. 1999 Feb 9; 1410(2):171–82. doi:10.1016/S0005-2728(98)00165-0.Brown A. M., et al.
Correlation of the clinical severity of Alzheimer’s disease with an aberration in mitochondrial DNA (mtDNA). J Mol Neurosci. 2001 Feb; 16(1):41–8. doi:10.1385 /JMN:16:1:41.Cavallucci V., Ferraina C., D’Amelio M.
Key role of mitochondria in Alzheimer’s disease synaptic dysfunction. Curr Pharm Des. 2013; 19(36):6440–50. Epub 2013 Feb 13.Chen J. X., Yan S. D.
Amyloid-beta-induced mitochondrial dysfunction. J Alzheimers Dis. 2007 Sep; 12(2):177–84. doi:10.3233/JAD-2007-12208.Duboff B., Feany M., Götz J.
Why size matters — balancing mitochondrial dynamics in Alzheimer’s disease. Trends Neurosci. 2013 Jun; 36(6):325–35. Epub 2013 Apr 11. doi:10.1016 /j.tins.2013.03.002.Gabuzda D., et al.
Inhibition of energy metabolism alters the processing of amyloid precursor protein and induces a potentially amyloidogenic derivative. J Biol Chem. 1994 May 6; 269(18):13623–8.Harman D.
A hypothesis on the pathogenesis of Alzheimer’s disease. Ann NY Acad Sci. 1996 Jun 15;786:152–68. doi:10.1111/j.1749–6632.1996. tb39059.x.Hu H., et al.
A mitocentric view of Alzheimer’s disease. Mol Neurobiol. 2016 Oct 1. Epub ahead of print. doi:10.1007/s12035-016-0117-7.Lustbader J. W., et al.
ABAD directly links Abeta to mitochondrial toxicity in Alzheimer’s disease. Science. 2004 Apr 16; 304(5669):448–52. doi:10.1126/science.1091230.Mariani C., et al.
Muscle biopsy in Alzheimer’s disease: morphological and biochemical findings. Clin Neuropathol. 1991 Jul; 10(4):171–6.Mark R. J., et al.
Amyloid b-peptide impairs glucose transport in hippocampal and cortical neurons: involvement of membrane lipid peroxidation. J Neurosci. 1997 Feb 1; 17(3): 1046–54.